UCL 1684 dibromide is a potent, non-peptidic blocker of small conductance calcium-activated potassium (SK) channels, exhibiting nanomolar potency. This compound demonstrates significant atrial-selective inhibition of sodium current (INa), effectively preventing the onset of atrial fibrillation. In addition, UCL 1684 dibromide induces atrial-selective prolongation of the effective refractory period (ERP) by promoting postrepolarization refractoriness, making it a valuable tool for researching cardiac electrophysiology and arrhythmogenesis.
UCL 1684 dibromide is a potent, non-peptidic blocker of small conductance calcium-activated potassium (SK) channels, exhibiting nanomolar potency. This compound demonstrates significant atrial-selective inhibition of sodium current (INa), effectively preventing the onset of atrial fibrillation. In addition, UCL 1684 dibromide induces atrial-selective prolongation of the effective refractory period (ERP) by promoting postrepolarization refractoriness, making it a valuable tool for researching cardiac electrophysiology and arrhythmogenesis.
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