MEG hemisulfate is a selective and potent inducible nitric oxide synthase (iNOS) inhibitor, exhibiting EC50 values of 11.5 μM for iNOS, alongside higher values for endothelial and brain nitric oxide synthases. In addition to its role as an iNOS inhibitor, MEG hemisulfate functions as a robust scavenger of peroxynitrite, mitigating peroxynitrite-induced oxidative damage. This compound demonstrates protective effects in various inflammation-related models, including ischemia/reperfusion injury, periodontitis, and inflammatory bowel disease, making it a valuable tool for research in inflammatory processes and oxidative stress responses.
MEG hemisulfate is a selective and potent inducible nitric oxide synthase (iNOS) inhibitor, exhibiting EC50 values of 11.5 μM for iNOS, alongside higher values for endothelial and brain nitric oxide synthases. In addition to its role as an iNOS inhibitor, MEG hemisulfate functions as a robust scavenger of peroxynitrite, mitigating peroxynitrite-induced oxidative damage. This compound demonstrates protective effects in various inflammation-related models, including ischemia/reperfusion injury, periodontitis, and inflammatory bowel disease, making it a valuable tool for research in inflammatory processes and oxidative stress responses.
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