Quin C1 is a potent and selective agonist of the formyl peptide receptor 2 (FPR2/ALX). This compound effectively reduces neutrophil and lymphocyte counts in bronchoalveolar lavage fluid (BALF) and diminishes the expression of pro-inflammatory cytokines such as TNF-α, IL-1β, KC, and TGF-β1, while also decreasing collagen deposition in lung tissue. Quin C1 is valuable for investigating mechanisms underlying lung injury and inflammation.
Quin C1 is a potent and selective agonist of the formyl peptide receptor 2 (FPR2/ALX). This compound effectively reduces neutrophil and lymphocyte counts in bronchoalveolar lavage fluid (BALF) and diminishes the expression of pro-inflammatory cytokines such as TNF-α, IL-1β, KC, and TGF-β1, while also decreasing collagen deposition in lung tissue. Quin C1 is valuable for investigating mechanisms underlying lung injury and inflammation.
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