Quercetin-d5, a deuterated form of Quercetin, serves as an autophagy inducer by modulating key signaling pathways. This natural flavonoid enhances the activity of recombinant SIRT1 and exhibits inhibitory effects on phosphoinositide 3-kinases (PI3K), with IC50 values of 2.4 μM for PI3K γ, 3.0 μM for PI3K δ, and 5.4 μM for PI3K β. Quercetin-d5 is utilized in research applications exploring autophagy modulation and the consequences on cellular metabolism and longevity.
Quercetin-d5, a deuterated form of Quercetin, serves as an autophagy inducer by modulating key signaling pathways. This natural flavonoid enhances the activity of recombinant SIRT1 and exhibits inhibitory effects on phosphoinositide 3-kinases (PI3K), with IC50 values of 2.4 μM for PI3K γ, 3.0 μM for PI3K δ, and 5.4 μM for PI3K β. Quercetin-d5 is utilized in research applications exploring autophagy modulation and the consequences on cellular metabolism and longevity.
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